Dear reader, please upgrade to the latest version of IE to have a better reading experience. Lawmakers during the joint session unanimously adopted a resolution on Kashmir and also passed the anti-rape and honour killing bills. Earlier this month, during the PTI's Raiwind march, PTI chief Imran Khan warned the ruling party he would not let the government function after Muharram if the premier did not present himself for accountability.
As Imran Khan announced the party's boycott of the joint session of parliament held last week, the PTI chief claimed the prime minister had two options. The other option is he can present himself for accountability on the Terms of Reference that the opposition has put forth," he said, adding that the premier had 'held the nation hostage' on this issue. It would have have happened surely if you being the custodian of the National Assembly would not be biased.
Imran Khan is always frustrated, always angry and always does idiotic things. No wonder he is always lacking support from majority of people in Pakistan. If He really wants to succeed in politics then he has to wash his brain first. The nation stands together on the issue of Kashmir but is not together on saving the people nominated in Panama Leaks and Lahore Model Town massacre.
Imran Khan's stance is percent correct. Wazir he may be all you say but he is true patriot.
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A major goal of biomedicine is to understand the function of every gene in the human being genome. Consanguineous unions are more likely to result in young who carry LoF mutations in a homozygous state.
In Pakistan, consanguinity rates are notably sybaritic. These pLoF mutations are predicted to knock out 1, genes in at least one into.
Finally, APOC3 is a gene which retards clearance of plasma triglyceride-rich lipoproteins and where heterozygous deficiency confers protection against coronary heart disease. Compared with wild-type family members, APOC3 knockouts displayed marked blunting of the old post-prandial rise in plasma triglycerides. Across all participants Table 1 , exome sequencing yielded 1,, exonic and splice-site sequence variants in 19, autosomal genes that passed initial quality control metrics. Of these, 57, mutations over 14, autosomal genes were annotated as pLoF mutations i.
Applying these criteria, we generated a set of 49, pLoF mutations across 13, autosomal genes. The site-frequency spectrum for these pLoF mutations revealed that the lion's share was seen only in anecdote or a few individuals Extended Data Fig. A full listing of all 1, genes knocked out, the number of knockout participants for each gene, and the specific pLoF mutation s are provided in Supplementary Pr�cis 1. Nearly 1 in 5 sequenced participants 1, individuals,
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- United States Eppig JT, et al. September 11, Zardari made friends of Pakistan: Single nucleotide polymorphisms SNVs and indel sites were initially filtered after variant calibration marked sites of low quality that were likely false positives. Christiansen D, et al. First, for genes where two or more participants were homozygous pLoF, we conducted an association screen against distinct phenotypes Supplementary Table 2.
Several limitations deserve mention. Age, sex, and myocardial infarction status were used as covariates in all analyses. PM Nawaz Sharif is relatively better than other rotten eggs. It must show patience and wait for court's verdict instead of disrupting the parliament activities. Arg19Ter proband and relatives Methods for Sanger sequencing We collected blood samples from a total of 28 subjects, including one of the four APOC3 p.
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